The N-myc and c-myc downstream pathways include the chromosome 17q genes nm23-H1 and nm23-H2

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Inhibition of a new differentiation pathway in neuroblastoma by copy number defects of N-myc, Cdc42, and nm23 genes.

The best studied oncogenic mechanisms are inactivating defects in both alleles of tumor suppressor genes and activating mutations in oncogenes. Chromosomal gains and losses are frequent in human tumors, but for many regions, like 1p36 and 17q in neuroblastoma, no mutated tumor suppressor genes or oncogenes were identified. Amplification of N-myc in neuroblastoma is strongly correlated with loss...

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Chemical intervention of the NM23-H2 transcriptional programme on c-MYC via a novel small molecule

c-MYC is an important oncogene that is considered as an effective target for anticancer therapy. Regulation of this gene's transcription is one avenue for c-MYC-targeting drug design. Direct binding to a transcription factor and generating the intervention of a transcriptional programme appears to be an effective way to modulate gene transcription. NM23-H2 is a transcription factor for c-MYC an...

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NM23-H1 and NM23-H2 messenger RNA abundance in human hepatocellular carcinoma.

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ژورنال

عنوان ژورنال: Oncogene

سال: 2002

ISSN: 0950-9232,1476-5594

DOI: 10.1038/sj.onc.1205259